Mitochondrial homeostasis in cellular models of Parkinson’s disease

Nina  Krako Jakovljevic; Brad  Ebanks; Gunjan Katyal; Lisa  Chakrabarti; Ivanka  Markovic; Nicoleta Moisoi

doi:10.26124/bec:2021-0002

Authors

Nina Krako Jakovljevic University of Belgrade, RS https://orcid.org/0000-0003-3908-5462
Brad Ebanks University of Nottingham, UK https://orcid.org/0000-0002-0067-430X
Gunjan Katyal University of Nottingham, UK https://orcid.org/0000-0001-8722-3343
Lisa Chakrabarti University of Nottingham, UK; MRC Versus Arthritis Centre for Musculoskeletal Ageing Research, UK https://orcid.org/0000-0001-7213-6837
Ivanka Markovic University of Belgrade, RS https://orcid.org/0000-0002-7961-3752
Nicoleta Moisoi De Montfort University, UK https://orcid.org/0000-0002-0400-9227

DOI:

https://doi.org/10.26124/bec:2021-0002

Keywords:

mitochondrial homeostasis, mitochondrial respiration, Parkinson’s disease, cellular models of Parkinson’s, qualitative analysis

Abstract

Mitochondrial function is known to be an important factor in maintaining cellular homeostasis and its dysregulation has become a hallmark for multiple disease conditions. This review aims to synthesise the extent of this knowledge by analysing changes of mitochondrial physiology parameters in Parkinson’s disease (PD) and to evaluate the contribution of cellular models of PD in the field. The analysis provided here constitutes a platform for further elucidation of mitochondrial function parameters relative to factors that may potentiate disease progression.

Cite:

Krako Jakovljevic N, Ebanks B, Katyal G, Chakrabarti L, Markovic I, Moisoi N (2021) Mitochondrial homeostasis in cellular models of Parkinson’s Disease. Bioenerg Commun 2021.2. https://doi.org/10.26124/bec:2021-0002

Mitochondrial homeostasis in cellular models of Parkinson’s disease

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